Deletion of Dtnbp1 in mice impairs threat memory consolidation and is associated with enhanced inhibitory drive in the amygdala

Keyphrases

• Amygdala 100%
• Basolateral Amygdala 50%
• Behavioral Deficits 16%
• Behavioral Phenotype 16%
• Brain Regions 16%
• Dendritic Spines 16%
• DTNBP1 100%
• Dysbindin 100%
• Dystrobrevin Binding Protein 1 16%
• Emotional Processing 16%
• Excitatory Neurons 16%
• GABAergic Inhibition 16%
• Heritable Disorders 16%
• Impact Function 16%
• Inhibitory Neurons 16%
• Male Mice 16%
• Memory Consolidation 100%
• Memory Expression 16%
• Morphological Abnormalities 16%
• MRNA Expression 16%
• Neuron Activity 16%
• Parvalbumin Interneurons 16%
• Pathophysiology of Schizophrenia 16%
• Patients with Schizophrenia 16%
• Protein Expression 16%
• Pyramidal Neurons 50%
• Schizophrenia 16%
• Schizophrenic Patients 16%
• Threat Memory 100%

Neuroscience

• Interneuron 14%